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Tiniazol (Ketoconazole)

Tiniazol is typically used as an antifungal medication used to treat fungal infections. However, it can be used in the treatment of advanced prostate cancer, although it is not approved by the FDA for this use. The information provided here focuses on the use of Tiniazol to treat prostate cancer. Tiniazol can inhibit the production of steroids in your body, including testosterone. The inhibition of the production of steroids can slow down and possibly stop the progression of prostate cancer.

Other names for this medication:
Ketoconazole, Nizoral, Akorazol, Arcolan, Arcolane, Asquam, Biogel, Clarazole, Conazol, Daktagold, Daktarin, Dezor, Diazon, Dikoven, Eumicel, Extina, Funginoc, Fungoral, Ilgem, Kenazol, Kenazole, Ketazol, Ketokonazol, Ketolef, Ketovid, Ketozal, Liondox, Lizovag, Micoral, Micosin, Muzoral, Mycoderm, Noell, Orifungal, Oronazol, Panfungol, Perative, Phytoral, Pristinex, Tiniazol, Tinuvin, Xolegel, Yucomy, Zoloral,

Similar Products:
Clindacin, Aclam, Tamsulosine, Clinda, Geodon, Locid, Vasilip, Diklofen, Reslin, Dhaperazine, Vonum, Nazee, Gabictal, Prazole, Biomox, Hiderax, Warlin, Bisoprolol, Corbis, Amoxysol, Ketovid, Clopilet, Renapril, Segan, Nasida,

See also Warning section. Tiniazol is used to treat certain serious fungal infections in the body. Tiniazol belongs to the class of drugs called azole antifungals. It works by stopping the growth of the fungus. Tiniazol should not be used to treat fungal infections in the brain or on the skin and nails. OTHER This section contains uses of this drug that are not listed in the approved professional labeling for the drug but that may be prescribed by your health care professional. Use this drug for a condition that is listed in this section only if it has been so prescribed by your health care professional. This drug may also be used to treat advanced prostate cancer.

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Expression of the mutated p53 tumor suppressor protein and its molecular and biochemical characterization in multidrug resistant MCF-7/Adr human breast cancer cells. Regulation of cysteine cathepsin expression by oxidative stress in the retinal pigment epithelium/choroid of the mouse. Extracted lymph nodes were surgically stratified into three distinct anatomical zones by two sagittal planes running in front of the aorta and the inferior vena cava.

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Knockdown of Minichromosome Maintenance Proteins Inhibits Foci Forming of Mediator of DNA-Damage Checkpoint 1 in Response to DNA Damage in Human Esophageal Squamous Cell Carcinoma TE-1 Cells. Light-microscopically, the tumor was composed mainly of nests of rhabdoid cells with fields of PNET. Our preliminary observations demonstrated the feasibility and efficacy of this novel surgical procedure as a promising option in T2DM.

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Poor physical performance is common in patients with kidney failure on dialysis (CKD-5D). SP-A and SP-D can interact with receptor molecules present on immune cells leading to enhanced microbial clearance and modulation of inflammation. Diagnostic significance of individual symptoms in formes frustes of acute dysentery in children

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shRNA-mediated suppression of DEP-1 in a colon epithelial cell line with high endogenous DEP-1 levels enhanced proliferation, further supporting the antiproliferative function of DEP-1. Ganglioside GM3 level per unit fresh weight and per unit fat-free-dry-matter was significantly higher in cold-acclimated rats (CA) than in warm controls, while the tissue triglyceride and glycogen levels were lower in CA. We propose that prostatein, a protein supplied by an accessory gland like prostate, can inhibit the macrophage function, showing an important antioxidant effect.

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